Semax

Core Functions

Enhances Cognition and Memory:
It specifically promotes the synthesis and release of neurotransmitters such as acetylcholine, dopamine, and norepinephrine in key brain regions such as the prefrontal cortex and hippocampus, enhancing signal transmission efficiency across synapses and significantly improving attention, working memory capacity, and information processing speed. Preclinical studies have shown its potential to improve age-related cognitive decline, mild cognitive impairment, and early symptoms of Alzheimer’s disease, particularly in improving memory consolidation and learning ability.

Anti-Fatigue and Stress Management:
By precisely regulating the overactivation of the hypothalamic-pituitary-adrenal axis, it inhibits excessive secretion of the stress hormone cortisol, reducing the damage to the nervous system caused by chronic stress. It also enhances energy metabolism, alleviates mental fatigue and physical fatigue, and improves psychological resilience and stress tolerance. It has a certain auxiliary effect on improving negative emotions such as anxiety and depression, as well as stress-related behavioral disorders. Neuroprotection and Repair:
In pathological conditions such as cerebral ischemia, brain trauma, and stroke, Semax can block neuronal apoptosis by inhibiting the activity of caspase family proteases. It also promotes the synthesis and secretion of nerve growth factor and brain-derived neurotrophic factor, accelerating axonal regeneration and synaptic remodeling in damaged neurons. Furthermore, it can scavenge free radicals and inhibit the release of proinflammatory factors, reducing secondary damage to neural tissue caused by oxidative stress and inflammation. This has important research value in functional rehabilitation treatment after neurological injury.
Improving Sleep Quality:
By regulating the balance of sleep-related neurotransmitters such as serotonin and gamma-aminobutyric acid in the brain, Semax helps stabilize the brain’s sleep-wake cycle, shortening sleep onset latency, reducing the number and duration of nighttime awakenings, and extending the duration of deep sleep. It has a particularly significant effect on insomnia and sleep fragmentation caused by chronic stress, neurological disorders, or excessive cognitive load.